Skip to main content

Author: Sam Visnic

Top 6 Calf Release Techniques

 

The calf muscle group involves 2 primary muscles: the gastrocnemius and the soleus.  Both of these muscle merge into the achilles tendon.

The most common reasons for working on the calves include plantar fasciitis pain, and the inability to achieve optimal depth in the squat.

Gastrocnemius
Soleus

How to know if you may need the following calves release techniques:

  • Anterior pelvic tilt
  • Challenges squatting deeply
  • Calf strain
  • Ankle or knee pain
  • Plantar fasciitis

Are Your Calves REALLY Tight?

How do you know if your calves are actually tight?

They can be tested with a goniometer passively (with a therapist), or actively with a knee reach test.  Both of these tests can also include testing ankle mobility.

Watch the video below to learn more about calf flexibility testing.

Calf Release – Massage Therapy
The calves are generally very responsive to massage therapy work, but can often be very tender. They appear to respond well to medium pressure and many repetitions. Over time, as tolerance improves, the pressure can be progressively increased.

Calf Stretching – Standing – Gastrocnemius Version

The standing calf stretch is the most common way to improve calf flexibility.  In particular, this standing version emphasizes the more superficial calf muscle, called the gastrocnemius, because the knee is straight.

It’s a common error for the heel to rise during this stretch, which can make it difficult to access the muscle, so always make sure to follow the steps to maximize the stretch.

VIDEO

Calf Stretching – Standing – Soleus Version

The standing calf stretch can not only target the larger gastrocnemius, but also the deeper soleus muscle.

The difference between these stretches is simple:  In order to access the soleus, the knee must be bent.  Many people find it difficult to really feel the soleus muscle stretching, usually feeling more ankle restriction than stretch.

If this is the case, you may find the kneeling ankle mobilization to be more useful.

Calf Release With Percussion Therapy Tool

Percussion tools can be helpful for freeing up tight calf muscles.  The easiest position in which to apply this technique is the half-kneeling.

This is additionally a good way to improve ankle mobility and squat depth.

VIDEO

Graston-IASTM Therapy Tool For Calves

Graston-type IASTM (instrument assisted soft tissue mobilization) tools can be helpful for improving calf flexibility and improving recovery.

These tools are mostly used by professional therapists, but the techniques can be safely implemented at home.

Foam Rolling Calves For Self-Massage

Self-massage for the calves is easily do-able with a standard foam roller.  The goal is to roll with a light-moderate level of discomfort that allows you to comfortably work on the tissues.

When you find areas that are particularly tender, hold those points for 10-15 seconds, then move to another area.

Continue this process until you treat the areas that you can access effectively.

VIDEO

 

Can Orthotics Help With Pain?

Biomechanics of the lower body are not as simple as they might appear.  There’s quite a bit of pressure involved in every step we take, and even if we take it for granted, our feet and knees are always subject to a lot of stress.

The good news is, we are built to handle this kind of stress.  There are some specific circumstances, however, that can cause us to need some extra help.  This is where orthotics may be helpful.

Orthotics is a speciality that focuses on creating devices called orthoses.  There are shaped according to the contours of your limbs or to stabilize, alter or reduce range of motion, depending on what they are being used for.

In foot orthoses, special inserts like arch supports, or even special shoes can be designed with the intent on changing how we walk or run.

All of this sounds very powerful in its potential to affect the body, and many people presume all sorts of physical issues may be improved by the use of orthotics for the feet, but does it really work this way?

How effective are orthotics really?

Do you really need orthotics?

The orthotics field is complex, and if we want to consider the relationship between orthotics and pain, we have to narrow down the focus to two different types of pain that are pretty common:  foot pain and lower back pain.

One of the most reliable sources of scientific info comes from Cochrane reviews, and one of them analyzed whether or not custom-made foot orthoses are helpful to address foot pain.

The authors concluded that there is very good evidence that they improve pain in SPECIFIC pathologies, such as pen cavus, plantar fasciitis, hallux valgus, and various types of arthritis.

If any of these aren’t present, according to their review, there is limited evidence for assigning custom-made foot orthotics (1).

What about back pain?  Since there is not that much evidence, this is where the controversy shows up.  If we look through the literature, some studies show that id does work to alleviate back pain.

However, after taking a looking further into the research, there appears to be a lack of real scientific arguments, while others report that orthotics for back pain have the same effect as placebo.

Despite the apparently reasonable-sounding claims by people who want to make the sale, there are 4 main issues that appear to benefit from orthotics, and a few others that might as well:

  • Plantar fasciitis:  This is essentially inflammation in the sole of the foot, around the heel and arch.  It’s quite painful in the morning, and is thought to be often caused by increased pressure over your plantar fascia ligaments (2,3).
  • Arthritis:  When arthritis affects the joints of the foot, orthotics might come in handy.  This is especially true in the case of things like deformative arthritis like rheumatoid arthritis (4).  Still, according to the literature, arthritis by itself is not an indication for orthotics, and they are often used when it’s not really necessary (5).
  • Metatarsalgia: In this case, an evaluation of the gait cycle and abnormalities of the foot may be needed to find the potential source of pain.  In some cases, it is associated with overloading abnormalities, and orthotics may come in handy and improve symptoms in both the short and long term (6,7).
  • Diabetes and other foot-related pathologies:  Pathologies that compromise circulation of the foot may require orthotics in order to reduce pressure on the planter foot and in the incidence of some complications, such as in the case of diabetes (8).

So, what if you do NOT have any of these issues?  Can orthotics help?

Well, it’s possible, but there isn’t much research to support the use of them in such cases.

What about orthotics to improve postural pain?

You may have noticed that back pain is not listed as a problem that requires the use of orthotics…

That’s because there’s no clear evidence that they can directly improve back pain issues!

You might be wondering, ok…but what about improving posture?  Doesn’t improved posture mean less pain?

Of course one of the most common arguments is that orthotics will improve posture as a result of changing gait, and as a result, back pain is resolved.

This depends greatly on WHY the postural issue exists.  (well beyond the scope of this blog post). In short, most back pain is classified as “non-specific”, and it’s often resistant to most forms of standard therapy.

This is because there’s most often many sources of input that contribute to the pain (speaking especially toward chronic back pain) and trying to address it with a pair of orthotics isn’t going to work (9).

So, even though poor posture, work ergonomics, etc. can contribute to pain, it’s often given way more importance than it deserves.

One area to look at is leg length differences.  It’s often thought that if you have a “short leg”, your gait will become irregular and you will end up with some type of physical pain, whether it be knees, hips, lower back, etc.

Studies show that assessments of leg length differences are likely to be biased, even in chiropractors with over 30 years of experience (10)!

Even if you do have a significant difference, it is clear that people with this problem do not have more back pain issues relative to everyone else (11).

Orthotics for lower back pain: Yay or Nay?

A recent systematic review and meta-analysis of several clinical trials on shoe insoles and its relationship with low back pain state that there is not enough evidence to support the use of insoles to treat or prevent low back pain (12).

Even though some studies claim that there’s a significant improvement after using orthotics (13), the truth is that after analyzing the methodology of their studies, there’s a statistical but not a clinical significance. In other words, even though their statistics might seem to indicate that there’s an improvement, it is not translated into real value in the clinical practice (14).

So to sum it up, orthotics appear to be most helpful when they are used for specific types of pain, mainly located in the foot and associated with overloading and pressure problems.

Since orthotics, especially when custom-made, can get pretty pricy, they clearly aren’t the go-to when it comes to other types of chronic aches and pains.

They certainly don’t have more direct value than hands-on massage techniques, exercises, stretches, and self-mobility drills.

Graded Exposure: The Key To Movement Success

Most health professionals recommend resting the area affected by an acute injury in order to recover.  In many cases, due to the extent of the injury, this may absolutely be necessary.  However, it appears that recently the tide is turning in terms of how soon movement is recommended in order to speed recovery from certain types of injury or even surgical procedures.

In fact, getting things moving again may reduce the risk of developing chronic pain.

Movement has different effects in the acute vs. chronic pain sufferer, but the process we are going to discuss in assigning it is focused on the chronic pain sufferer.  This concept is called graded exposure.

Pain itself can be warning sign that protects us from re-injury, but sometimes people adopt an undesired pain-avoidance behavior that does more harm than good.  The concept of graded exposure has been designed with these individuals in mind.

Fear of movement and re-injury

Pain and common disabilities associated with it are influenced by many different things, and some of them do not appear to respond to a structural-biomechanical approach.  Psychological and psychosocial factors play an important part, and individuals with fearful thoughts related to movement often experience more pain and disability in the long run.

It’s also known that longstanding avoidance of movement and compensatory strategies (altered movement due to previous injury or fear of movement) may lead to a progressive impairment of movement skill.

Thus, we need to differentiate pain disability and the disability that results from something like a musculoskeletal problem.  Sometimes, musculoskeletal problems are gone, injuries are healed, but the disability remains due to fear of movement and avoidance behavior.

It appears that often health professionals that work with individuals that suffer from chronic pain tend to forget is that perhaps the primary obstacle that is being experienced isn’t the tissue itself, but the fear of performing certain activities or movements because the individual believes they are re-injuring the painful area.

What is graded exposure?

Graded exposure is that concept that is based on progressively exposing individuals to increasing levels of stress in order to stimulate adaptation in the central nervous system.

It can significantly reduce sensitivity to movement and avoidance behaviors that contribute to chronic pain.

What needs to be considering in graded exposure is applying just the right amount of stress that will not cause any setbacks in progress, but instead stimulate adaptation.

This principle is easily applied to areas such as muscle building, for example.  Every workout session, the stress increases just a bit more, either through more repetitions or added weight.  Doing too much hampers progress, and may lead to injury.

We can easily apply this concept to virtually all therapeutic modalities, including exercise, stretching, massage therapy, neuromuscular techniques (such as muscle energy techniques), and much more.

Is there a difference between graded exposure and graded exercise?

There are many applications of graded exposure, and we should differentiate this to graded exercise.  Graded exposure is a concept of progressively increasing stress to overcome the fear of movement and modulate the nervous system, but as mentioned, can be applied to many different types of therapeutic modalities.

Graded exercise is a common meth0d that is meant to teach an individual that they can do certain movements and reach certain ranges of motion via a progressive approach.  This is especially helpful for individuals that suffer from chronic pain and are afraid to perform certain movements or activities.

As it is, graded exposure is part of graded exercise, and it’s certainly possible to apply the principles of both at the same time.

 

Graded exposure and adaptation of the nervous system

Several studies have identified a strong link between pain perception and mood.  Anger and negative emotions lower the pain threshold, making us more sensitive to pain, and giving us extra reasons to feel irritable.

Thus, it is widely accepted that our psychological and social circumstances modulate our interpretation of pain, and our reactivity and levels of vigilance.  Our perception of safety is also important, and this is what progressively changes during graded exposure.

Achieving a dosage of stress that triggers very low-intensity pain with a controlled movement is away to send a signal to your nervous system that this move or particular range of motion is safe to perform.

In time, it will feel more comfortable and less pain will occur with this movement because you will have adapted to this stressor.  More stress can be applied to create further adaptation, and reduce the pain “alarm” further as your nervous system becomes de-sensitized to the stimulus.

This technique is generally based on mainly observational and anecdotal evidence, but there is also scientific literature that establishes the foundations and the role of graded exposure in cases of pain.

Still, there is no final guideline or definitive recommendation to guide the application of graded exercise in particular.  This is where the art of the work and experience comes into play.

There are some principles that are helpful to follow:

  • Assessing fear of movement:  There are many potential ways to assess fear of movement.  From graded tests to simple client interview and demonstration.
  • Implementation of movements and stress loads:  Sometimes there are multiple movements that are problematic, and/or activities that don’t produce discomfort until load is applied.  These can be divided up and dealt with one at a time.  Improvements in some movements may directly improve others without specifically addressing them!
  • Re-evaluation and modifications:  After a given time that depends on the individual’s situation (often times within the same visit), the problematic movements or tests can be repeated, and adjustments made to further improve the response.

The bottom line is that graded exposure and exercise can be an extremely valuable component of any chronic pain sufferer’s movement and/or massage therapy program.

The power lies in identifying activities, movements, ranges of motion, and other factors which trigger fear, or avoidance, and creating a plan to systematically reduce or eliminate that fear by starting with education, then selecting an appropriate low-threatening way to begin, and progressing over time as results are attained.

 

References:

Vlaeyen, J. W., Kole-Snijders, A. M., Rotteveel, A. M., Ruesink, R., & Heuts, P. H. (1995). The role of fear of movement/(re) injury in pain disability. Journal of occupational rehabilitation, 5(4), 235-252.

Tuna, Z., & Oskay, D. (2018). Fear of movement and its effects on hand function after tendon repair. Hand surgery and rehabilitation, 37(4), 247-251.

George, S. Z., & Zeppieri Jr, G. (2009). Physical therapy utilization of graded exposure for patients with low back pain. journal of orthopaedic & sports physical therapy, 39(7), 496-505.

Can Lifting Weights Give You Relief From Pain?

There are few things in the health field as versatile as exercise.  It is commonly recommended and sometimes prescribed a s part of the therapy for various health conditions.

Even though we assume this type of recommendation for people with excess weight and even cardiovascular issues, it is also very helpful for people that suffer from chronic pain.

First, before diving into this topic, we need to separate exercise into two main types:  aerobic and anaerobic.  Aerobic exercise is also known as cardiovascular (cardio), and anaerobic exercise is  known as resistance training, weight training, or strength training.

Below, we’ll evaluate what the research shows when it comes to the effective use of anaerobic training to relieve chronic pain.

Do pain symptoms improve with resistance training?

While many studies show that resistance training improves pain symptoms, other show it might not be all that useful.  This can definitely seem confusing, but this discrepancy can exist for a few potential reasons, including the type of pain that is being experienced, and of course the WAY the training is being performed.

If you have a properly designed program and you’re doing everything correctly, you’ll likely to get improvement from some types of chronic pain (1).

A recent review and meta-analysis of resistance training and chronic lower back pain show that aerobic exercise and resistance training were both associated with a significant reduction in pain intensity. However, resistance exercise displayed an added benefit to the psychological well-being of the individual, which is essential to modulate the perception of pain. (2).

It’s also interesting to note that there are many variants of resistance training, and each of them has proven to reduce pain in a different way.  For instance, we can divide resistance training into static and dynamic muscle contractions.

Static muscle contraction, which includes isometric exercise (muscle contraction with no movement), is associated with a moderate-to-large modulation of pain, and certain studies have found that maintaining contraction at a low weight for a longer time recruits and exhausts more muscle fibers, leading to a more significant exercise-induced-hypoalgesia.

On the other hand, dynamic resistance, which is performed essentially by shortening and lengthening the muscles, has been found to have similar effects, but usually in the short-term (3).

When and how is strength training appropriate for chronic pain?

Resistance training can be utilized for various types of chronic pain, including fibromyalgia, which is known to be a fairly challenging condition.

Fibromyalgia is a pain condition that is highly variable between people that have it, and it’s associated ultimately with a dysfunction of the nervous system and increased susceptibility to pain experience.

Most of the information out there shows that controlling pain in fibromyalgia is an extremely challenging venture.  Even so, “old-school” resistance training is still an effective way to reduce the sensation of pain and tenderness, and maintain or increase muscle strength in these individuals.

According to a Cochrane review, aerobic exercise is superior to moderate intensity resistance training in some cases, so it appears to be a good idea to combine them to some degree (4).

One of the more common applications of resistance training in chronic pain is associated with things like repetitive strain injuries.  For example, a randomized control trial focused on strength training to treat working populations that had repetitive strain injuries, some of which had a work disability.

They underwent 10 weeks of strength training against the usual ergonomic care that is recommended to address this kind of pain. Strength training showed significant improvements in hand/wrist pain and reduced time to fatigue by an impressive 97% (5).

Achieving these improvements in chronic pain with resistance exercise is not so difficult. All we need is to choose key exercises per muscle group, include one or two sets for each exercise, choose the appropriate weight to perform a correct technique without a problem, and resting between sets a few minutes.

Each repetition should be performed at medium-speed, and doing this twice every week for each muscle group is usually enough for good results. This is in accordance with the current recommendations of physical activity for health by the World Health Organization, which includes 150 minutes of moderate physical activity (aerobic exercise) a week combined with muscle-strengthening activities 2 days a week or more (6).

Common obstacles and risks of resistance training

Generally in order to be effective, resistance training needs to be done 2-3 times per week.  This sounds easy, but there are some obstacles to be aware of.

  • You may not feel immediate improvements:  Even though there’s a connection between resistance training and pain sensitivity, it does not produce rapid shifts in pain perception like modalities such as massage or relaxation training.  Real improvements in this area take time, as much as 12 weeks to see the true benefits.
  • It’s about changing your mindset and lifestyle:  Resistance training will require time commitment and persistence.  If you’re not committed or lack the ability to stick to it, you aren’t likely to get the outcome you are seeking.

Resistance training may not be the thing that everyone needs for their type of chronic pain, or it may not fit into their needs at a particular time.

Here are a few more things to be aware of:

  •  Strength-training injuries:  Hitting the weights without any professional help, and no mastery of exercise technique may not only reduce the benefits, but also get you injured.  Getting injured in the process of trying to relieve pain is a common issue, and this is why you should seek help from a professional!
  • Imprecise exercise protocols for the type of pain:  Certain types of chronic pain won’t respond well to aggressive resistance training.  Some, like fibromyalgia, need to be very closely monitored because the training program may constantly need to be adjusted based on how the person is feeling.  Frozen shoulder is another issue that may not respond well to strength training at all, and in fact may make it much worse.

Summary

Resistance training (anaerobic exercise) certainly has its place in the therapeutic regime of the chronic pain sufferer and has been shown to be highly effective, but it must be applied to the right kind of pain issue, in the right way, and progressed over time as the person adapts to the program.

Is Fascia Really Relevant To Chronic Pain Relief?

From myofascial release, to fascial stretching therapy and fascial release tools, everyone seems to be focusing on this specific body tissue.

In this article, we’re going to take a look at fascia, as it’s been a rising hot topic in the alternative therapy realm, and whether or not it therapies targeted at fascia have value for the chronic pain sufferer.

What IS fascia?

In gross anatomy, fascia is simply connective tissue that wraps around muscles and other anatomical structures.  If is flexible and capable of stretching in response to muscle contractions and body movements, and it covers almost every organ, not only muscle groups.

So it IS flexible enough to move along with muscle so that it won’t obstruct its normal contraction.

The growing interest in fascia started by the idea that musculoskeletal pain is triggered by deformation of this connective tissue wrapping.  This model of pain has been named the “Fascial Distortion Model”, and was introduced in 1991 by Stephen Typaldos, but other therapists have talked about fascia in relationship to pain and musculoskeletal dysfunction over the years.  Therapists such as John Barnes have been teaching myofascial release for decades.

According to the Fascial Distortion Model, it is possible to fix fascial distortion by a series of techniques involving handwork and specialized tools.

Even though it is mentioned by the American Fascial Distortion Model Association that this model has proven useful in treating acute and chronic injuries, there is no scientific validation that such distortions actually exist or that manual therapy specifically can do anything about it.

Facts about fascia you need to know

Misconceptions about fascia and how it is related to muscle pain are EVERYWHERE now, and even skilled physical therapists, chiropractors, fitness trainers, and massage therapists are lured into fascination by its assumed implications.

Somehow we are led to believe that fascial restrictions can be identified through chains or patterns throughout the body, and targeted with either stretching, hands-on techniques, or using metal tools designed to work directly with fascia.

The problem is these claims are simply not well supported by science, and most research appears to show we can’t do much about fascia directly, especially not in the short-term.

This appears even more evident when we learn about the actual strength of fascia.  It’s pretty hard to believe any changes in extremely dense and tough tissue can be accomplished within the context of a therapy session, or even a few for that matter.

Consider the following facts:

It is NOT possible to change or deform fascia using manual therapy

A study published in the Journal of the American Osteopathic Association evaluated the ability to deform or change fascia via massage therapy.  This is relevant to the claim that fascial release improves pain because according to this model, therapists should be able to stretch or fix “fascial deformations” in the first place.

However, the evidence pointed out by this paper shows that mechanical deformation and stretching of fascia is only possible by using high forces (on the plantar fascia and IT band), while certain types of fascia (in the nose for example), may be influenced by less.

To put this in perspective, in order to deform the fascia on the bottom of the foot (plantar fascia) by 1%, you would need to apply more than 8,000N of force.   The force of a lion’s jaw can produce a bit more than 4,000N of force!

This is because this thin layer of connective tissue is too tough to lengthen by simple common clinical therapeutic techniques.  The amount of pressure needed to do so would be extremely stressful (if even possible) by the therapist, and certainly be extremely painful to the recipient.

Thus, it’s not likely at all that these alleged fascial distortions can be fixed by massage, or stretching techniques.

Fascial contraction and pain

Some studies show that similar to the muscles they cover, fascia can undergo a weak and slow type of contraction.  This is undoubtedly exciting information and destroy the thought that fascia is form of inert connective tissue.

However, it should be stressed that contractions found in fascia are extremely subtle and difficult to register, especially when you compare them to that of muscles.

The question is whether or not there is any real significance to these contractions, especially in the context of therapeutic techniques. (Actual practice, not just in a test tube!)

Is it possible that such a weak contraction can influence the correct functioning of muscle groups?

That’s a tough sell to assume that these weak contractions generated in-vitro would act by themselves in the body causing deformations to the fascia and triggering pain.

Another entertaining thought is that any manual therapy practitioner can actually assess or feel the release of fascial tissues, and be able to distinguish them from both the muscles contained, as well as all of the tissues that lay on top of them! Virtually impossible.

We haven’t even touched on the issue that pain itself is not purely a structural issue.  We know that tissue damage and pain are not fully associated, so even if damage to fascia is found through deformations, etc., it doesn’t mean it has ANYTHING to do with pain!

The primary mechanism for pain relief lies in changes to your nervous system

I’ve already mentioned how tough the fascial covering of a muscle is and that is very unlikely to mold or fix fascia with manual therapy.

Doing so would require excessive pressure that is inconceivable in massage or stretch therapy.

However, there is definitely something that feels different after these heavy-pressured massage or stretching sessions, and of course many people report feeling pain relief.

Does this prove that fascia needs to be released?  Does it validate the narrative often espoused by these fascia-focused therapies?

No, it doesn’t prove that at all.  There are a great many potential reasons why people feel better after hands-on therapy sessions.

Endorphins improve pain, novel sensory input can alter perception of pain, activating the parasympathetic aspect of the nervous system, and even the placebo effect are all potential explanations and common effects from manual therapy.

The take home point here is that we can emphasize the end result of feeling better through movement and manual therapy techniques, and get rid of the likely false story of WHY those techniques are working.

One of those false stories is that you feel better because of changes to your fascia, and how it could have been altered as an effect of the therapy.

 

References:

Thalhamer, C. (2018). A fundamental critique of the fascial distortion model and its application in clinical practice. Journal of bodywork and movement therapies, 22(1), 112-117.

Chaudhry, H., Schleip, R., Ji, Z., Bukiet, B., Maney, M., & Findley, T. (2008). Three-dimensional mathematical model for deformation of human fasciae in manual therapy. The Journal of the American Osteopathic Association, 108(8), 379-390.

Schleip, R. (2003). Fascial plasticity–a new neurobiological explanation: Part 1. Journal of Bodywork and movement therapies, 7(1), 11-19.

Meltzer, K. R., Cao, T. V., Schad, J. F., King, H., Stoll, S. T., & Standley, P. R. (2010). In vitro modeling of repetitive motion injury and myofascial release. Journal of bodywork and movement therapies, 14(2), 162-171.

Schleip, R., Klingler, W., & Lehmann-Horn, F. (2006). Fascia is able to contract in a smooth muscle-like manner and thereby influence musculoskeletal mechanics. Journal of Biomechanics, 39(1), S488.

https://www.nationalgeographic.com/news/2012/3/120315-crocodiles-bite-force-erickson-science-plos-one-strongest/

Massage Myths: Can You Really Break Up Scar Tissue, Knots, And Adhesions?

Massage therapy has been around for a VERY long time, and recent science has backed up many arguments that were formerly enveloped in mystery regarding its practice. However, there are still myths and misconceptions to address, especially since many of them are still being perpetuated by health professionals as part of their social media marketing efforts.

The concept of breaking up “knots”, “adhesions”, and scar tissue is one in particular.  One of the most popular questions people seek answers for is how to massage scar tissue to break it down.

Marketing vs. Reality

Since there is no way to monitor every single event and physiological change during an actual massage session, scientists need to take facts and draw conclusions accordingly. This makes massage therapy more susceptible than many other health practices to myths and misconceptions based on subjective and personal experiences.

Thus, it is pretty common to see professional therapists continue to recommend debunked practices like drinking more water after a massage session to clear “toxins” away or using vague terminology like “freeing up scar tissue and adhesions”, “releasing fascia“, or “digging out knots”.

According to some bodyworkers, adhesions are formed by dysfunctional fibrous tissue that attaches to the muscle, prevents it from stretching, restricts the range of motion of the body, and causes muscle pain. I specifically recall being taught these things when I first became a massage therapist.

In school we were taught that these knots were mis-aligned muscle fibers that rearrange in bunches that become hypersensitive and tense.  It’s not uncommon to see topics like these all over the internet from massage therapists:

  • How to break up scar tissue
  • How to break up fascia
  • How to break up fascial adhesions at home
  • Massage for adhesions
  • Scar tissue massage therapy

These concepts have become so widespread in people that even massage therapists who do not believe in them (I’m one of them!), are forced to use the language as part of the discussion with the person in order to assess their situation and convey the outcome of their therapeutic program!

There is, however, a long distance between this widely used marketing babble and what science has really found out about the musculoskeletal system.

What are knots, adhesions, and scar tissue really?

If you look up the term “adhesion” in a medical-type textbook, the only reference you will find is to a type of scar tissue that is commonly associated with inflammation, and is capable of sticking neighboring tissues together.

These adhesions are a common outcome of surgery, and the only way to release them is by cutting through with a scalpel. You won’t find any other usage of the word “adhesion” associated to muscles or surrounding tissues spontaneously gluing together, as is often implied by many bodyworkers, and there’re no scientific literature on the best practices to release them by using touch therapies.

Thus, scar tissue is not likely to create adhesions without having had surgery. They are mostly post-interventional problems, except in some instances. For example, females with a prolonged history of pelvic pain may create scar tissue and adhesions around the uterus. Definitely not associated to muscle issues, and certainly can’t be resolved with massage!

What about muscle “knots”? There’s at least SOME truth to defend this concept because muscle tension feels different than a relaxed muscle, and it is not something that untrained individuals may realize. Even considering, this doesn’t mean that the muscle fibers have somehow become distorted, or its collagen fibers become mis-aligned. Due to this, there is nothing to “re-align” with massage!

The trigger point debate continues to rage on…

When researching about muscle knots, it is highly likely you will come across terms such as trigger points and myofascial pain.  If you look at the concept of trigger points, in essence it’s the same as what has been described as a muscle knot.

A trigger point is said to be a hypersensitive muscle area that is tense and usually feels like a bump or palpable nodule.  In some cases, pressing this area would elicit pain local to the area, or radiate to another location.

Whether or not trigger points are real is a cause of ongoing debate.  What people FEEL during or after a massage isn’t on trial, but the explanations for pain and discomfort are.

Trigger point supporters say muscle stress affects blood circulation and promotes the accumulation of metabolic waste in this painful area.  The area becomes acidic, stimulates nerve endings, and this contributes to tightening up muscle tissue even more in a vicious cycle.

While this explanation sounds reasonable, critics highlight that there’s no scientific proof to back up these arguments, and it is no more than pure speculation.

Moreover, therapy based on the idea of trigger points have the same results as placebo therapy, contributing to the argument that the concept of trigger points is no more than a wild guess.

So, CAN massage break up adhesions, scar tissue, and muscle knots?

Whether or not muscle knots actually exit, massage therapy contributes to improving pain associated with this type of ache.  However, it is not because of a chemical or mechanical dysregulation of the muscle tissue.  It’s a result of changes in the nervous system and skillful touch by a massage therapist that contributes to soothing muscle tension in these areas.

But, what about using more force and performing a super deep tissue, painful massage in order to break up adhesions, scar tissue, and release muscle knots?

This concept is very similar to what is known as tissue provocation therapy, a type of healing technique that is based on how tissues become able to adapt to stress.  According to this type of treatment, we can force adaptation in the affected tissue by applying stress.

For example, a Chinese massage technique called Gua sha uses hard tools to apply pressure and achieve maximum penetration to break up scar tissue, muscle adhesions, fascial restrictions, and other alleged musculoskeletal problems.

According to safety studies, this technique is associated with severe skin burns, dermatitis, and other nasty side effects.

Unfortunately, there is also no magical massager to break up scar tissue, no matter how fancy the tool looks.

So what do we make of the “muscle knots popping sound” that is heard when experiencing massage?

Muscle tendons can flip over bones as they move when massaged, and joints cavitate (pop) as when getting a chiropractic adjustment.  While it isn’t always clear to answer the why do muscles pop during massage question, it’s certainly not something like “toxins being released”, as commonly stated by many massage therapists.

Provocation therapy is unnecessarily painful and has the same effects as a placebo therapy.  This only shows how people look for exotic treatments with impressive claims to raise expectations, even more if they are painful and someone with authority claims it’s a necessary step for recovery.

Nope.  Inducing pain is not required to achieve what really matters, which is a change in your nervous system that will contribute to relaxing your muscles, improving neuronal chemistry, and alleviating pain!

 

References:

Hall, H. (2010). The Graston Technique–Inducing Microtrauma with Instruments. sciencebasedmedicine. org. Retrieved, 01-09.

Cheatham, S. W., Lee, M., Cain, M., & Baker, R. (2016). The efficacy of instrument assisted soft tissue mobilization: a systematic review. The Journal of the Canadian Chiropractic Association60(3), 200.

Vashi, N. A., Patzelt, N., Wirya, S., Maymone, M. B., Zancanaro, P., & Kundu, R. V. (2018). Dermatoses caused by cultural practices: Therapeutic cultural practices. Journal of the American Academy of Dermatology79(1), 1-16.

Schleip, R. (2003). Fascial plasticity–a new neurobiological explanation: Part 1. Journal of Bodywork and movement therapies7(1), 11-19.

Hutson, M., & Ward, A. (Eds.). (2015). Oxford textbook of musculoskeletal medicine. Oxford University Press.

Is Aerobic Exercise Helpful For Chronic Pain?

Is Aerobic Exercised Helpful For Chronic PainExercise is often recommended for many different health conditions, especially those related to the cardiovascular system, but it’s certainly not the only area where it may be extremely valuable.

Living a sedentary lifestyle is a fairly significant risk factor for health issues, and it not only limited to stroke and heart disease.

Chronic pain is included in the list of reasons to recommend physical activity, and even though many people are apprehensive of being more active when they have pain, studies show that it works and improves symptoms both in the short and long-term.

There are two main forms of physical activity (aerobic and anaerobic), and each of them is associated with an improvement in chronic pain.

We’re going to focus on aerobic exercise (often referred to as “cardio”), and how pain mechanisms can be inhibited by this simple form of movement.

What happens when you do aerobic exercise?

During aerobic exercise, the most significant changes involve the respiration and cardiovascular systems.  Your muscles play an essential part, and the stroke volume of your heart increases to make up for the increased requirement of oxygen and nutrients.

Thus, the function of your capillaries is enhanced, and the part of your cells that produces energy, called mitochondria, increases in both number and efficiency.

Additionally, there are many nervous system and hormonal adaptations that occur after aerobic exercise.  These changes improve both strength and stamina, and have the ability to reduce insulin resistance and other metabolic problems.

Aerobic exercise can also play a role in improving flexibility, balance, coordination, and also positively impact muscle/postural asymmetry.

All of these improvements can be associated to various pain inhibitory mechanisms we’ll discuss below.

How does aerobic exercise inhibit pain?

The exact mechanism by which physical activity improves chronic pain symptoms is not entirely understood.  Unfortunately the vast majority of clinical trials do not make a clear distinction between aerobic and anaerobic physical activity when examining the effects of exercise on pain perception.

Most of these studies are a combination of interventions, therapeutic recommendations, and patient education, which makes it even more difficult to set aside and evaluate the differences between aerobic and anaerobic exercise separately.

That said, it is still possible to make a distinction based on what we do know by looking at the short and long-term effects of aerobic exercise.

Short-term pain inhibition from aerobic exercise

Reduction of pain perception, called hypoalgesia, has been reported after physical activity in healthy individuals, people with chronic pain, and experimentally-induced pain.

There are two mechanisms involved in this short-term numbing effect: one of them is the release of endorphins, and the other is a modulation in the pain pathways in the central nervous system.

According to studies, after high-intensity aerobic exercise, there’s a hypoalgesic (numbing) effect lasting approximately 30 minutes.  This effect is longer compared to resistance training exercise (anaerobic exercise), which lasts only a few minutes.

Studies seem to show that more exercise or a higher intensity is related to more hyperalgesia.  The suggestion is that to generate a considerable effect we should engage in high-intensity physical activity for more than 10 minutes or moderate to high physical activity for 30 minutes or more.

Now of course you can imagine this wouldn’t work in a number of scenarios.  For example some clinical trials have reported an INCREASED pain response instead of pain inhibition in cases of individuals with severe chronic pain.

People with fibromyalgia, for example, do not have immediate pain inhibition after physical activity, but of course they can still benefit from exercise in the long run.

Long-term pain inhibition from aerobic exercise

The long-term pain inhibitory effects of aerobic exercise are only reported in individuals that maintained a regular program of activity.

Long-term activity is known to modulate the endocannabinoid system (natural cannabis-like molecules produced by the human body), reduce the inflammation by improving circulation, and normalize transmission of glutamate in the brain and pyruvate in the muscle tissue.

By altering the release of inflammatory substances and improving circulation, it is possible to reduce swelling and other symptoms that may be associated with some chronic pain issues.

The endocannabinoid system in particular has a strong inhibitory effect that’s commonly targeted by pain medications.  This system is also modulated by aerobic activity after releasing endorphins.

Additionally, glutamate and pyruvate are essential for sensory perception in the muscle tissue and its transmission in the central nervous system.  Thus, by reducing these substances, physical activity may modulate pain perception in the long-run.

Psychological effect of exercise

We can’t talk about pain perception without including the psychological function.  It is well established that depression, anxiety, and similar mental health problems have a negative impact on pain perception.

Individuals that engage in aerobic exercise usually report a significant improvement in mood and psychological symptoms, which may have a positive effect on the chronic pain experience.  There are not, however, enough studies to evaluate these changes in a large amount of people suffering from chronic pain.

So, does aerobic exercise help?

In a nutshell, aerobic exercise is known to reduce pain perception immediately after exercise and in the long-run.  However, for long-lasting results, it is necessary to engage in sustained activity, and most cases of chronic pain would also benefit from combining some aerobic exercise with anaerobic exercise.

By joining the strengthening nature of anaerobic exercise and the physiological changes mentioned in this article, it is possible to reduce pain symptoms and improve quality of life for chronic pain sufferers.

 

References:

Kawi, J., Lukkahatai, N., Inouye, J., Thomason, D., & Connelly, K. (2016). Effects of exercise on select biomarkers and associated outcomes in chronic pain conditions: systematic review. Biological research for nursing18(2), 147-159.

Naugle, K. M., Fillingim, R. B., & Riley III, J. L. (2012). A meta-analytic review of the hypoalgesic effects of exercise. The Journal of pain13(12), 1139-1150.

Nijs, J., Kosek, E., Van Oosterwijck, J., & Meeus, M. (2012). Dysfunctional endogenous analgesia during exercise in patients with chronic pain: to exercise or not to exercise?. Pain physician15(3S), ES205-ES213.

Bender, T., Nagy, G., Barna, I., Tefner, I., Kádas, É., & Géher, P. (2007). The effect of physical therapy on beta-endorphin levels. European journal of applied physiology100(4), 371-382.

Koltyn, K. F. (2002). Exercise-induced hypoalgesia and intensity of exercise. Sports medicine32(8), 477-487.

Sajedi, H., & Bas, M. (2016). The evaluation of the aerobic exercise effects on pain tolerance. Sport Science9, 7-11.

Naugle, K. M., & RILEY 3rd, J. L. (2014). Self-reported physical activity predicts pain inhibitory and facilitatory function. Medicine and science in sports and exercise46(3), 622.

Gerdle, B., Ernberg, M., Mannerkorpi, K., Larsson, B., Kosek, E., Christidis, N., & Ghafouri, B. (2016). Increased interstitial concentrations of glutamate and pyruvate in vastus lateralis of women with fibromyalgia syndrome are normalized after an exercise intervention–a case-control study. PloS one11(10), e0162010.

Ghafouri, N., Ghafouri, B., Fowler, C. J., Larsson, B., Turkina, M. V., Karlsson, L., & Gerdle, B. (2014). Effects of two different specific neck exercise interventions on palmitoylethanolamide and stearoylethanolamide concentrations in the interstitium of the trapezius muscle in women with chronic neck shoulder pain. Pain Medicine15(8), 1379-1389.

Your Sleep Issues May Be Making You Hurt

Sleep is essential for life, and every organism with a nervous system requires this type of resting phase. Moreover, it is noted that other types of organisms without a brain do have a circadian rhythm as well.

This only highlights the importance of sleep in the physiology of living beings. It is further noticed that sleep deprivation is associated with a number of diseases, including mental health problems, an impairment in the immune system, and metabolic dysfunction including obesity and type 2 diabetes.

But what about chronic pain?

Is there any connection between sleep deprivation and chronic pain?

What relevant scientific findings can you apply in your day-to-day life to improve your symptoms of chronic pain?

Is there a link between pain and sleep deprivation?

The connection between chronic pain and sleep deprivation has been established for a long time. For instance, back in 1979, it was noted in a scientific study that rats who were subject to sleep deprivation had a significantly lower pain threshold, which means they are more susceptible to pain.

More recent studies in humans have confirmed the validity of these findings, and it has been found to be a risk factor to suffer recurrent migraine attacks. Similarly, the current evidence on the matter describes a higher pain perception in various settings, including fibromyalgia, joint dysfunction, and muscle soreness.

The majority of studies agree that not having enough sleep at night predisposes people to chronic pain, increases the severity of the symptoms, and the recurrence of the attacks because it is associated with hyperalgesic changes.

In other words, when you don’t sleep properly, your body starts feeling pain differently. This might be proven in different patients with different sources of pain, as discussed in the following section.

How sleep modulates pain in various settings

Besides clinical trials on migraine, there is plenty of supporting evidence to show a definite link between sleep fragmentation or deprivation and chronic pain. We can see the scientific evidence in various medical settings:

  • Arthritis and joint pain: According to a clinical trial published in 2016, sleep fragmentation induces an increased sensitization to joint pain. The study compared patients with knee arthritis with a healthy sleep pattern with others who did not sleep properly. After this comparison, the authors concluded that treating sleep is fundamental to improve chronic pain in patients with arthritis.
  • Fibromyalgia: This is a condition that features chronic pain, and it is often difficult to trace and diagnose. According to a survey performed in patients with this type of pain, insomnia was strongly associated with pain (not as a result of pain but as an aggravating factor).
  • Low back pain: It is a common problem, and most people have at least one episode throughout their lifetime. However, it was found that 53% of people with chronic symptoms of low back pain have sleep problems as compared to 3% of pain-free patients who reported insomnia.
  • Other musculoskeletal problems: One of the first associations between sleep deprivation and pain was around the issue of musculoskeletal symptoms. Sleep-deprived patients report an increase in muscle tenderness and more frequent musculoskeletal symptoms. Moreover, recent findings pointed out that the perception of musculoskeletal pain in these patients is increased by 24%.

All of this evidence points out that people who do not sleep properly have a higher sensitivity to pain. However, a learned individual may wonder if there’s a real organic change or is it only a subjective sensation.

In other words, is pain more severe or is the patient emotionally biased to feel more pain? According to the evidence published in the journal Psychosomatic Medicine, pain sensitivity is increased, it is possible to measure this organic change, and there’s no additional alteration in perceptions that may contribute to bias.

The most recent discoveries about pain and sleep deprivation

Even though there are many studies on the human brain, and the connection between sleep and chronic pain has been defined for so long, there were no studies on brain perception of pain during periods of sleep deprivation until recently.

In January 2019 it was discovered that healthy young people who do not sleep properly have higher activity in brain areas that trigger pain (the primary somatosensory cortex) and reduced activity in brain areas that coordinate movement and modulate our perception of pain (the insular cortex and the striatum).

After this study, we can trace the exact reason why sleep deprivation modulates pain. For some reason still to be explained by subsequent studies, not having proper or sufficient sleep increases the sensitivity of the sensory network, primarily focusing on neurons that trigger pain.

At the same time, the body becomes unable to modulate or mute pain sensation and perceives more sharply these sensory impulses. Moreover, the study found that even mild sleep deprivation affects pain perception.

These studies and medical knowledge have a suitable therapeutic potential in a hospital setting and highlights how important it is for patients suffering from chronic pain to have proper sleep at night.

Similarly, it should be a wakeup call for anyone who is continuously disrupting their sleep pattern and feeling any type of pain symptom the day after. Insomnia hurts, and there’s much we can do about it.

One of the most severe chronic pain diseases is fibromyalgia, and according to a clinical trial, applying a few sleep hygiene tips has been found to improve the symptoms in these patients.

Recommendations include sleeping every day at the same hour, not using the same room to sleep, eating and studying, turning off screens before going to sleep and avoiding coffee, alcohol, tobacco and heavy, greasy foods.

There are many ways to improve our quality of sleep and reduce our perception of chronic pain. What we have to do is raise our awareness of the importance of sleep and follow easy recommendations.

References:

Hicks, R. A., Coleman, D. D., Ferrante, F., Sahatjian, M., & Hawkins, J. (1979). Pain thresholds in rats during recovery from REM sleep deprivation. Perceptual and motor skills, 48(3), 687-690.

Alstadhaug, K., Salvesen, R., & Bekkelund, S. (2007). Insomnia and circadian variation of attacks in episodic migraine. Headache: The Journal of Head and Face Pain, 47(8), 1184-1188.

Orlandi, A. C., Ventura, C., Gallinaro, A. L., Costa, R. A., & Lage, L. V. (2012). Improvement in pain, fatigue, and subjective sleep quality through sleep hygiene tips in patients with fibromyalgia. Revista brasileira de reumatologia, 52(5), 672-678.

Burton, E., Campbell, C., Robinson, M., Bounds, S., Buenaver, L., & Smith, M. (2016). Sleep mediates the relationship between central sensitization and clinical pain. The Journal of Pain, 17(4), S56.Tang, N. K., Wright, K. J., & Salkovskis, P. M. (2007). Prevalence and correlates of clinical insomnia co‐occurring with chronic back pain. Journal of sleep research, 16(1), 85-95.

Jansson, C., Mittendorfer-Rutz, E., & Alexanderson, K. (2012). Sickness absence because of musculoskeletal diagnoses and risk of all-cause and cause-specific mortality: a nationwide Swedish cohort study. PAIN®, 153(5), 998-1005.

Moldofsky, H., & Scarisbrick, P. (1976). Induction of neurasthenic musculoskeletal pain syndrome by selective sleep stage deprivation. Psychosomatic medicine.

Lentz, M. J., Landis, C. A., Rothermel, J. S. H. A. V. E. R. J. L., & Shaver, J. L. (1999). Effects of selective slow wave sleep disruption on musculoskeletal pain and fatigue in middle aged women. The Journal of rheumatology, 26(7), 1586-1592.

Caffeine and Chronic Pain: Are They Linked?

Caffeine and Chronic Pain CoffeeCaffeine has been a staple in the human diet for generations, and its social and cultural significance far exceeds that of most other psychoactive substances.

Avid consumers of caffeine report that it keeps them alert, active, and improves their outlook.  This, and the fact that caffeine is legal and widely available, makes it into the world’s most widely used drug.

For many individuals, caffeine is part of the daily diet, and in some cases, its consumption may become excessive.  For this reason, we need to be aware of the effects of caffeine, and how it may impact people with chronic pain.

How do you take your caffeine?

Caffeine is included in a variety of beverages and foods, but by far the most commonly consumed is coffee, followed closely by sodas, teas, and energy drinks.

What does caffeine DO to the body?

Caffeine exerts various effects on the human body, primarily enhancing alertness and prolonging attention, and it does this through direct stimulation of the central nervous system. This is why a cup of coffee can make it much easier to focus on tasks that are often boring!

The stimulant action of caffeine occurs due to its action as an adenosine antagonist and adenosine receptor agonist.  Through this action, it plays a role in the regulation of brain activity as well as the states of wakefulness and sleep.

This has caused many scientists to become interested in the many ways in which caffeine consumption may affect human health.  The subject is highly controversial, especially with regard to the relationship that exists between caffeine and chronic pain.

Caffeine is added to pain medications?

Because of all its reported health benefits, caffeine has been added to a variety of drugs over the years.

The primary formulations being aspirin, acetaminophen, and other non-steroidal anti-inflammatory drugs (ibuprofen).  Caffeine was likely originally added in order to offset the latent sedative effects of some of these drugs.

However, many now view caffeine as a substance that accelerates, prolongs, or enhances the analgesic effects of these pain relief medications.

How much caffeine is too much?

Before we get into the murky waters of discussing caffeine’s interaction with pain, we need to address the issue of how much caffeine is considered normal vs. excessive.

Due to the rapid and demanding pace of modern life, caffeine consumption is increasing around the world.  People of all ages are studying or working more and sleeping less, and caffeine presents an effective alternative to combat the effects of fatigue and exhaustion.  Many individuals are actually unaware of the amount of caffeine they consume on a daily basis and often fall into overconsumption.

Given that the Wold Health Organization recognizes caffeine dependence as a clinical disorder, it’s an important question to ask, how much is too much?

For the most part, caffeine’s characteristics make it a substance whose use has a high safety margin.  Caffeine absorption after oral administration is rapid and thorough.  It is distributed in most body tissues and crosses the blood-brain barrier very easily, which explains its potent stimulant effects.

Most of the world’s health agencies recognize that low to moderate doses of caffeine present with no complications on health.

It is important to note that if consumption of caffeine is done in a self-administered manner (without medical prescription), it is recommended to not exceed a daily dose of 400 mg.  To put that into perspective, consider that a 16 oz. cup of Starbucks Blonde roast packs about 360 mg!

Considering how widely available coffee shops are and the social aspects of consuming coffee with co-workers, friends, and family, it’s easy to see how consuming over the recommended daily ceiling of 400 mg is possible.

Excessive caffeine intake and chronic pain

The relationship between caffeine and pain lies a bit on the complicated side and deserves more scrutiny.

Case in point, habitual excessive caffeine consumption has been repeatedly associated with the appearance of headaches.  It may also interfere with the analgesic action of some pain-blocking agents, according to recent studies.

Excessive caffeine intake can have the following effects:

  • It may decrease the pain threshold and increase susceptibility to pain by activating the stress response.
  • It may cause muscle spasms, muscle twitching, and muscle tension, all of which are clinically significant components in individuals with chronic pain.
  • It can generate physical dependence, which can manifest as a withdrawal syndrome if intake is abruptly stopped.
  • Additionally, there is a psychological component that needs to be addressed.  Caffeine in excess can increase anxiety in even healthy individuals.  Since people dealing with chronic pain are often already experiencing increased levels of anxiety, stress, and anger, caffeine may very well increase pain by increasing the person’s susceptibility and awareness of it.

The chronic effects of caffeine intake are based on an up-regulation of certain types of receptors (adenosine) as well as their hypersensitivity.  Thus, caffeine has the ability to make chronic pain worse. For some individuals, too much caffeine (or any at all for some people) may be a recipe for disaster.

The caffeine and headache connection

Of particular note is the relationship between caffeine intake and the development of medication overuse headaches.

Headaches caused by the excessive use of pain relievers is a type of chronic headache, which occurs in susceptible individuals.  It’s considered a serious condition and now stands as the third most common type of chronic headaches around the world.

Therefore, when caffeine is consumed excessively for ongoing periods of time, it may very well induce distinct neurological changes that increase cortical hyperexcitability and promote the rapid release of certain neuropeptides, which can trigger or exacerbate the chronicity of pain.

Caffeine, sleep and chronic pain

The majority of studies agree that not having enough sleep or having poor quality of sleep predisposes people to chronic pain, increases the severity of the symptoms, and the recurrence of the attacks because it is associated with hyperalgesic changes.  In other words, when you don’t sleep properly, your body starts feeling pain differently.

It’s well known that excess caffeine intake, or consuming it too late in the day can negatively impact one’s ability to get to sleep.  This reduces total sleep time, thus increasing the likelihood of creating more problems with pain sensitivity.

The verdict:

Caffeine has both pluses and minuses, and as with most things, it’s not absolutely good or bad, but needs to be evaluated in the case of the individual. 

When caffeine is consumed in the form of coffee in particular, there are many additional potential benefits which need to be considered as well.  Maybe some decaf instead of full strength! 

 

References:

Sawynok, J. (2011). Caffeine and pain. Pain152(4), 726-729.

McPartland, J. M., & Mitchell, J. A. (1997). Caffeine and chronic back pain. Archives of physical medicine and rehabilitation78(1), 61-63.

Pectoral Muscle Strength Technique: Top 6 Pec Stretch Release Techniques

Power and flexibility often go hand in hand.

To maximize pectoral muscle strength techniques, it’s important to have the right level of mobility. Not only will this help you perfect new techniques, but it’s also going to help stave off injuries.

When it comes to strengthening the pectorals, these six pec stretch release techniques can unlock even more potential.

What Is Pectoral Muscle Release?

The pectoral muscle group consists of 2 muscles:  the pec major and the pec minor.

These muscles can often become tight, leading to excessively rounded shoulders, shoulder or neck pain, and breathing imbalance. Pec stretch release techniques can help alleviate these pains, restoring greater mobility and allowing the patient to focus on their pectoral muscle strength technique.

As well as helping with shoulder and neck problems, the pec stretch release techniques shown below can be effective for improving many issues.

Pec Release Techniques

When Might You Need Pectoral Muscle Release (Pec Major and Pec Minor):

Your pecs are important muscles in your body, so it’s important to maintain flexibility in this area, particularly if you’re working on pectoral muscle strength techniques.

However, there are some tell-tale signs that you might need pectoral muscle release:

  • Shoulder pain
  • Rounded shoulders
  • Neck pain
  • Referred pain down the arms
  • Imbalanced breathing or rib cage patterns

Movement is an important part of recovery, and in these cases, the right pec stretches can help alleviate pain and return mobility to the affected area.

Pec Muscle Release – Massage Therapy

The pectoralis major is the most superficial and easy to access chest muscle.  It starts at the sternum and inserts into the upper arm.  There are a variety of glides that can be used as demonstrated in the video, and even simple compression of the muscle can be used to assist with reducing tender areas.

The pectoralis minor, however, is a bit more challenging to access because it lies underneath the larger pec fibers.  It needs to be treated from the side, or side-lying position.

With treatment from an experienced professional and the right stretching routine, it is possible to release the pectorals, allowing people to get back to the workouts they love.

6 Pec Stretch Release Techniques

Stretching out the pecs is an important part of working on your pectoral muscle strength technique. The more mobility you have in this area, the better quality workouts you can do, leading to more effective and healthy muscle growth.

Pec Major Stretch – Standing Wall Stretch

One of the easiest and most familiar ways to get a pectoralis major stretch is with a doorway stretch.  Place your arm so that your elbow is slightly higher than your shoulder and step forward a bit while simultaneously turning your body away from the arm.

One thing to note, if the anterior shoulder capsule is sensitive for any number of reasons (underlying shoulder pathology) you may feel discomfort instead of the intended effect which is to stretch pectoralis major.  In this case you may need to opt for a self-massage style technique, or seek additional help to address the shoulder issue prior to doing these stretches.

Pec Stretch – Swiss Ball Stretch

The swiss ball pectoral stretch is a good alternative to the doorway stretch, especially if you have a hard time getting a good stretch.

Make sure you use a ball that is large enough, and that you allow your shoulder blades to move toward each other to simulate stepping forward through the doorway as discussed in the previous stretch.

Pec Muscle Release – Post Isometric Relaxation

Post-isometric relaxation works well for the pectoral muscles.

This is accomplished by moving toward the end of a massage table (or bench) and allow your arm to hang as if you were doing a stretch with gravity assistance.  If you are doing this by yourself, lift up your arm (using your chest muscles) just enough to activate the muscle.

Hold for 8-10 seconds, take a deep breathe in, and exhale as you attempt to relax further into the stretch.  Hold for 10 seconds, then repeat 2-3 more times or until no additional range of motion is achieved.

Pec Minor Release – Massage Therapy

Massage therapy works very well for the pec minor muscle.

Correct positioning as  well and low-moderate pressure is ideal for addressing this muscle.  You don’t need to push too hard!  In addition, it’s important to be cautious of nerves in the area of the pec minor.

Pec Major/Minor Muscle Release – Self Massage w Ball

Self-massage for the pectoral muscles is possible with the use of a tennis or lacrosse ball.  Place the ball into the desired area and lean into a wall or doorway.

You may wish to move your arm slightly back behind your body to lengthen the pectorals as you compress the tissues with the ball.  You can move your body to scroll through the tissues, and isolate any areas that are particularly tender.  It is fairly common to find pectoralis major trigger points that refer to the shoulder and often down the arm.  Hold those points for 10-15 seconds, then move to another area.

Continue this process until you treat the areas that you can access effectively.

Conclusion

Stretching should be an essential part of your pectoral muscle strength technique. If you’re suffering from pain in your neck and shoulders, then it’s going to hold back your workout, and it could be due to your pectorals.

With the right treatment, and some pec release techniques, you can alleviate pain and make your workouts even more effective.

Mobility is key and pectoral muscle release could be exactly what you need.